I've found some relevant comments regarding my questions on this Patreon page (behind the paywall and in Polish, but I've machine translated some small interesting bits):This is an interesting case: First case of monkeypox virus, SARS-CoV-2 and HIV co-infection.
Isn't that a very good ground for recombinations? I mean, the fact that the patient is immuno-suppressed and got all those three viruses detectable (with symptoms). Or maybe coinfections like this are common?
(...) In turn, the MPXV virus can acquire features from the SARS2 RNA hybrids if some RNA-dependent DNA polymerase in the cell is active. Such a polymerase is ... reverse transcriptase, originating from HIV. It synthesizes DNA based on RNA.
So, we are just basically waiting for something bad to happen. The comments also confirm that MPXV can also acquire characteristics from spike protein encoded in the vaxes:(...) creating a situation in which different viruses of the same and other types will be capable of co-infection uninhibited by the natural mechanisms of the immune system - will generate a whole range of quasi-viruses with completely new features and thus the route of infection and symptoms. It is nothing more than a reshuffle of the entire genetic pool of all viruses that can and infect humans, and the main mechanism behind it are mmRNA vaccines acting as a trigger - "facilitator" of simultaneous occurrence of previously strictly controlled molecular mechanisms of RNA and DNA replication.
(...) It is also possible that if MPXV recombination occurs with the genetic material of the SARS2 S protein, it will be particularly efficient in people who have been vaccinated with adenovector