Mass Extinctions, Evolutionary Leaps, and the Virus-Information Connection

Michael B-C

FOTCM Member
Pierre. Just to say your book arrived a couple of days ago. I'm only a few chapters in, so can't give a thorough response yet, but thus far thoroughly enjoying the read. As ever you distill so much information into an immediate yet clear and economical fashion that I find myself utterly gripped. What a page turner! I just love the way you introduce the reader to what would normally be mind-blowing deductions (e.g. water exchange between earth and Mars) without the slightest sense that we should be surprised or in anyway find this outlandish. The whole argument holds together like super-glue and I look forward to learning much from your work - as always - and enjoying the roller-coaster ride to the end! Much respect. Thank you and all the supporting team for all the undoubted effort that went into its distillation and publication.


FOTCM Member
Pierre. Just to say your book arrived a couple of days ago. I'm only a few chapters in, so can't give a thorough response yet, but thus far thoroughly enjoying the read. As ever you distill so much information into an immediate yet clear and economical fashion that I find myself utterly gripped. What a page turner! I just love the way you introduce the reader to what would normally be mind-blowing deductions (e.g. water exchange between earth and Mars) without the slightest sense that we should be surprised or in anyway find this outlandish. The whole argument holds together like super-glue and I look forward to learning much from your work - as always - and enjoying the roller-coaster ride to the end! Much respect. Thank you and all the supporting team for all the undoubted effort that went into its distillation and publication.
Wow, what a nice comment! :clap:
I'm really glad you enjoy your reading. If you have a few minutes to spare, you could adapt the above quote into a review on Amazon, positive reviews are our best advertising.


A Disturbance in the Force
To give an idea of the structure of the book and its lines of force, here's the temporary table of content:

Introduction : 4

Part 1 : comets and mass extinction 13
Mass extinctions 14
Causes of mass extinction 16
The 27 million year cometary cycle 22
Other cometary induced mass extinctions 32

Part II : Life explosion 36
Saltarism vs Darwinism 37
Peculiar life forms around cometary impacts 39
The K/T boundary 43
The cambrian life explosion 45
Other life explosion 46
Beyond the mechanical action of cometary event 49

Part III: Viruses are the drivers of life 54
Anteriority of viruses 55
Pervasiveness of virus 57
Beneficial viruses 58

Part IV: Viruses and meteorites 62
Comet and plagues, an historical perspective 63
Meteorites carrying micro organism 65
Upper atmosphere microorganism 70
Electro Magnetic pulses 72

Part V: The information Field 77
Information theory & consciousness 78
The Field 84
Collective resonance 94

Part VI: Information field, viruses and protein 99
DNA as antennas 100
Proteins as tuners 102
DNA and proteins connect us to the Information Field 106
Fractals 108
From geometric shape to electromagnetic signature 112
The interesting case of homeopathy 116

Part VII: 6th extinction and coronavirus 120
The ongoing 6th extinction 121
Authorities and Life 123
Authorities and Control 126
Dumbing Down the Population 128
The Mind-Altering Virus 130
Man-Made Viruses 131
Man-Made SARS-CoV-2 132
Engineered for What Purpose? 135
Fort Detrick And Viruses 137
Fort Detrick and Mind Control 140
Made in China or Made in the USA? 142
From an Obedience Vaccine to a Mutated Disobedience Virus? 146
Covid vaccine 152
Conclusion 157

As you can see, Part I et II deal with mass extinctions and the subsequent evolutionary leaps. It shows that cometary events are the main cause of mass extinctions and that, soon after, more complex life forms seems to appear out of nowhere.

Part III and IV show that new viruses introduced by cometary events participate to remove obsolete life-forms and introduce the genetic material code enabling the evolutionary leap of the "spared" life forms.

Part V and VI show how the virus-enhanced DNA of life forms and the proteins coded by this DNA act as receivers tuned onto the information field. The properties of a substance are not defined by its composition but its geometry. The connection between proteins/DNA and the information field are geometric and electromagnetic. The fractal nature of DNA, viruses, proteins and the background ELF electromagnetic field reinforces the connection between life forms and any given "area" of the information field.

Part VII address the coronavirus story, an unstable Fort Detrick obedience virus that mutated in a disobedience virus (SARS-COV-2) for which obedience RNA vaccines had to be imposed. The weakening of the population immune system through fear, stress, no herd immunity and the deleterious RNA vaccines makes us highly vulnerable to a real epidemics (a new virus from space and/or a recombination between RNA vaccine sequences and viruses like the Black Death already contained in our DNA). From this perspective the 6th extinction might very well be man-made but not for the usually invoked reason: global warming. However, like during the preceding mass extinctions, the new viruses that remove obsolete life-forms might provide the genetic material for the survivors to experience an evolutionary leap.
Is it recommended to read the first book prior to this?
I love that you are addressing that viruses are an essential part of life and fundamental to our existence. Will you cover the working relationship between viruses and the innate immune system?
You mention some of the causes of the weakening of our immune system. Could you cover them all? Eg air pollution including glyphosates and pesticides in agriculture, aluminium plus other in chem trails from weather re-engineering, the fracking industry which releases cyanide into the atmosphere, radiation including emf’s wifi etc, all pharmaceutical products that are not natural and vaccines which have included mercury and aluminium plus a whole lot of crap not meant for the human body.
It would be great if this were a wake up call that the only health care we need is to build a robust immune system.
Someone somewhere must be measuring the air pollution levels but it needs to be a topic for discussion.
Thanks for the great work. I look forward to reading it.


FOTCM Member
You mention some of the causes of the weakening of our immune system. Could you cover them all?
Probably not all, there are so many! But there will probably be a chapter dedicated to EM pollution and how it interacts with DNA.

Is it recommended to read the first book prior to this?
Yes it is better to read the first book (Earth Changes and the Human-Cosmic Connection) because it introduces concepts like dynastic cycle, information field, electromagnetism that are used in this third book.
It is also better to read the second book (Cometary Encounters) because it introduces ideas like the correlation between cometary events, mass volcanism, earthquakes that are also used in this third book.


The Living Force
FOTCM Member
As I was doing my news round today I came across this article that reminded me of the thesis of your book Pierre.

Extinction at the end-Cretaceous and the origin of modern Neotropical rainforests

Another paper just came out showing how plants underwent two major diversification phases in dramatic bursts, 250 million years apart. The first major change happened in the early Devonian between 420 and 360 million years ago with the development of seeds and the second more dramatic change happened in the late Cretaceous where plants developed flowers.

Plants evolved complexity in two bursts—with a 250-million-year hiatus

A Stanford-led study reveals that rather than evolving gradually over hundreds of millions of years, land plants underwent major diversification in two dramatic bursts, 250 million years apart. The first occurred early in plant history, giving rise to the development of seeds, and the second took place during the diversification of flowering plants.

The research uses a novel but simple metric to classify plant complexity based on the arrangement and number of basic parts in their reproductive structures. While scientists have long assumed that plants became more complex with the advent of seeds and flowers, the new findings, published Sept. 17 in Science, offer insight to the timing and magnitude of those changes.

"The most surprising thing is this kind of stasis, this plateau in complexity after the initial evolution of seeds and then the total change that happened when flowering plants started diversifying," said lead study author Andrew Leslie, an assistant professor of geological sciences at Stanford's School of Earth, Energy & Environmental Sciences (Stanford Earth). "The reproductive structures look different in all these plants, but they all have about the same number of parts during that stasis."

An unusual comparison

Flowers are more diverse than every other group of plants, producing colors, smells and shapes that nourish animals and delight the senses. They are also intricate: petals, anthers and pistils interweave in precise arrangements to lure pollinators and trick them into spreading pollen from one flower to another.

This complexity makes it difficult for scientists to compare flowering plants to plants with simpler reproductivesystems, such as ferns or some conifers. As a result, botanists have long focused on characteristics within family groups and typically study evolution in non-flowering plants separately from their more intricate flowering relatives.

Leslie and his co-authors overcame these differences by designing a system that classifies the number of different kinds of parts in reproductive structures based on observation alone. Each species was scored according to how many types of parts it has and the degree to which it exhibited clustering of those parts. They categorized about 1,300 land plant species from about 420 million years ago until the present.

"This tells a pretty simple story about plant reproductive evolution in terms of form and function: The more functions the plants have and the more specific they are, the more parts they have," Leslie said. "It's a useful way of thinking about broad-scale changes encompassing the whole of plant history."

From shrubs to blooms

When land plants first diversified in the early Devonian about 420 million to 360 million years ago, Earth was a warmer world devoid of trees or terrestrial vertebrate animals. Arachnids like scorpions and mites roamed the land amongst short, patchy plants and the tallest land organism was a 20-foot fungus resembling a tree trunk. After the Devonian, huge changes occurred in the animal kingdom: Land animals evolved to have large body sizes and more varied diets, insects diversified, dinosaurs appeared—but plants didn't see a major change in reproductive complexity until they developed flowers.

"Insect pollination and animal seed dispersal may have appeared as early as 300 million years ago, but it's not until the last 100 million years that these really intricate interactions with pollinators are driving this super high complexity in flowering plants," Leslie said. "There was such a long period of time where plants could have interacted with insects in the way that flowering plants do now, but they didn't to the same degree of intricacy."

In the Late Cretaceous, about 100 to 66 million years ago
, Earth more closely resembled the planet we know today—a bit like Yosemite National Park without the flowering trees and bushes. The second burst of complexity was more dramatic than the first, emphasizing the unique nature of flowering plants, according to Leslie. That period gave rise to plants like the passionflower, which can have 20 different types of parts, more than twice the number found in non-flowering plants.

The researchers classified 472 living species, part of which Leslie carried out on and around Stanford's campus by simply pulling apart local plants and counting their reproductive organs. The analysis includes vascular land plants—everything except mosses and a few early plants that lack supportive tissue for conducting water and minerals.

"One thing we argue in this paper is that this classification simply reflects their functional diversity," Leslie said. "They basically split up their labor in order to be more efficient at doing what they needed to do."


Dagobah Resident
Hi Pierre,
I've taken some notes and links from some of the video lectures I've taken the time to watch (Thread ).
I haven't had time to dig deeper and connect the dots yet, but I thought that instead of keeping them until then, they might be useful to you in some way.

"Endogenous retroviruses (ERVs) are genetic parasites of vertebrate genomes. They are the 'footprints' of ancestral retrovirus infections that have integrated their genome into the germline of a host and then been transmitted vertically from generation to generation. Following germline colonisation, these elements can evolve in different ways. Some VRE can proliferate in the DNA of their host, resulting in the formation of multicopy families and the invasion of the genome of entire populations. Other elements may progressively degenerate and lose their ability to multiply, remaining only as fossil sequences in the host genome. Finally, some VREs may be retained, wholly or partially, by positive selection pressure if they provide a benefit to their host in terms of evolutionary success. This evolutionary diversity has resulted in a wide variety of VRE repertoires in different vertebrate species."

"Schizophrenia and bipolar disorder are neuropsychiatric disorders of unknown origin. It seems that these two disorders share some common etiopathogenic mechanisms including genetic, environmental and inflammatory ones. Reactivation of the human endogenous retrovirus type W (HERV-W) can be a shared element in the pathophysiology of schizophrenia and bipolar disorder, linked to immuno-genetic and environment risk factors. We will present studies that have highlighted the presence of HERV-W in schizophrenic and bipolar disorder patients. We will then describe a two-hit model which could explain the common pathophysiological mechanism of affective and non-affective psychosis. Identification of immuno-inflammatory mediated subgroup of schizophrenia and bipolar disorder associated to HERV-W reactivation might open the way for the development of diagnostic biomarker and more targeted treatments. These new tools pave the way towards personalized psychiatry for a better care of patients."
Les rétrovirus endogènes humains, une implication dans la schizophrénie et le trouble bipolaire | médecine/sciences


"Mammalian genomes contain a heavy load of retroelements, which are mobile sequences requiring reverse transcription for their amplification. A significant proportion of these elements is of retroviral origin, with thousands of sequences resembling the integrated form of infectious retroviruses with two LTRs bordering internal regions homologous to the gag, pol, and env genes. These elements, named endogenous retroviruses (ERVs), are thought to be the remnants of ancestral germline infections by active retroviruses, which have thereafter been transmitted in a Mendelian manner. The sequencing of several mammalian genomes has allowed a comprehensive study of their ERVs. They can be grouped according to sequence homologies into 10-100 families per genome, each containing a few to several hundred elements. Strong similarities between ERVs and present-day retroviruses can be inferred from phylogenetic analyses performed on the pol or env genes, suggesting a common history. As a general rule, most ERVs are old and degenerated, with their open reading frames disrupted, but a few proviruses have retained intact genes and the corresponding proteins can thus be expressed. Some elements still contain gag and pol genes that drive the synthesis of viral particles, as well as envelope genes whose product can be incorporated on their cognate or heterologous viral particles. This presentation will review the general properties of endogenous retroviruses, in relation with their possible consequences on vaccine production."
Risks linked to endogenous retroviruses for vaccine production: a general overview - PubMed

Prolonged SARS-CoV-2 RNA shedding and recurrence of PCR-positive tests have been
widely reported in patients after recovery, yet these patients most commonly are non-infectious.
Here we investigated the possibility that SARS-CoV-2 RNAs can be reverse-transcribed and
integrated into the human genome and that transcription of the integrated sequences might
account for PCR-positive tests. In support of this hypothesis, we found chimeric transcripts
consisting of viral fused to cellular sequences in published data sets of SARS-CoV-2 infected
cultured cells and primary cells of patients, consistent with the transcription of viral sequences
integrated into the genome. To experimentally corroborate the possibility of viral retro-
integration, we describe evidence that SARS-CoV-2 RNAs can be reverse transcribed in human
cells by reverse transcriptase (RT) from LINE-1 elements or by HIV-1 RT, and that these DNA
sequences can be integrated into the cell genome and subsequently be transcribed. Human
endogenous LINE-1 expression was induced upon SARS-CoV-2 infection or by cytokine
exposure in cultured cells, suggesting a molecular mechanism for SARS-CoV-2 retro-integration
in patients. This novel feature of SARS-CoV-2 infection may explain why patients can continue
to produce viral RNA after recovery and suggests a new aspect of RNA virus replication.

Beljanski discovers transforming RNAs. Excreted by mutant strains of Escherichi a coli , they are capable of transforming wild-type bacteria of the same species: they acquire the distinctive characteristics of the mutants and in turn excrete the transforming RNA.

The transforming RNAs are small and exceptionally rich in purine bases (G and A). They are synthesised by the PNPase, independently of the DNA, and normally remain 'stuck' to it: they also exist, in this inactive state, in wild-type bacteria. Under special circumstances, they can detach from the DNA and become active; this is what happens in mutants under the influence of the antibiotic showdomycin.

These RNAs can also transform bacteria of a different species from the one from which they originate. When they are taken up by a plant-cancer-causing bacterium, Agrobacterium tumefaciens, stable transformed strains appear, some of which have lost their tumorigenic potential.

The transformed strains no longer contain any trace of the transforming RNA. The transforming RNA has therefore transferred hereditary information from one bacterial strain to another. For the first time in the history of biology, proof has been provided that an RNA can cause a stable and heritable genetic transformation.

"Over the past decade, a large body of work has highlighted an ever-increasing number of RNA molecules that, unlike mRNAs, are not directly involved in the of RNA molecules that, unlike mRNAs, are not directly involved in protein synthesis.
protein synthesis. In other words, these RNAs are devoid of genetic information capable of coding for proteins. They are referred to as non-coding RNAs (ncRNAs) whose repertoire complexity equals or exceeds that of mRNAs.
repertoire equals or exceeds that of mRNAs. Although the biological function of the vast majority of ncRNAs remains largely enigmatic, some of them function as regulators of the flow of genetic information by controlling the
genetic information flow by controlling chromatin structure, regulation of transcription, mRNA stability and stability of mRNAs and the production and activity of proteins.

MicroRNAs and C/D snoRNAs are among the best characterised ncRNAs to date. These small ncRNAs selectively interact with other RNAs via base pairing and modify their
modify their reactivities. MicroRNAs block the translation of target mRNAs, whereas snoRNAs C/D snoRNAs guide the formation of chemical modifications (addition of methyl groups) at specific
positions along the target RNAs. The research activity of our team concerns a subclass of microRNAs and C/D snoRNAs whose gene expression defies the classical laws of heredity. Indeed, although the genes of these small ncRNAs are present in two copies in each nucleus (one allele inherited from the father, the other from the mother), only one of the two copies is active, and this strictly according to its parental origin. For example, for a given gene it is always the copy of paternal origin that is active, whereas the copy of maternal origin, which may be genetically identical, is silenced. There is therefore a "cellular memory" capable of distinguishing the parental origin of the alleles but also of modifying their activity. Such epigenetic phenomena are grouped under the term parental genomic imprinting.
In this talk, I will summarise our understanding of the production and mode of action of these small atypical ncRNAs, including a description of work that aims to elucidate the
physiological function of many microRNAs through the study of a knock-out mouse model. Finally, I will Finally, I will discuss the hypothesis that deregulations in the expression of certain C/D snoRNAs snoRNAs contribute to the etiology of a rare human disease: Prader-Willi syndrome."

We are writing to express concern about the use of a recombinant adenovirus type-5 (Ad5) vector for a COVID-19 phase 1 vaccine study,and subsequent advanced trials. Over a decade ago, we completed the Step and Phambili phase 2b studies that evaluated an Ad5 vectored HIV-1 vaccine administered in three immunisations for efficacy against HIV-1 acquisition. Both international studies found an increased risk of HIV-1 acquisition among vaccinated men.
The Step trial found that men who were Ad5 seropositive and uncircumcised on entry into the trial were at elevated risk of HIV-1 acquisition during the first 18 months of follow-up.
The hazard ratios were particularly high among men who were uncircumcised and Ad5 seropositive, and who reported unprotected insertive anal sex with a partner who was HIV-1 seropositive or had unknown serostatus at baseline, suggesting the potential for increased risk of penile acquisition of HIV-1. Importantly for considering the potential use of Ad5 vectors for COVID-19 infection, a similar increased risk of HIV infection was also observed in heterosexual men who enrolled in the Phambili study.
This effect appeared to persist over time. Both studies involved an Ad5 construct that did not have the HIV-1 envelope. In another HIV study, done only in men who were Ad5 seronegative and circumcised, a DNA prime followed by an Ad5 vector were used, in which both constructs contained the HIV-1 envelope.No increased risk of HIV infection was noted. A consensus conference about Ad5 vectors held in 2013 and sponsored by the National Institutes of Health indicated the most probable explanation for these differences related to the potential counterbalancing effects of envelope immune responses in mitigating the effects of the Ad5 vector on HIV-1 acquisition.The conclusion of this consensus conference warned that non-HIV vaccine trials that used similar vectors in areas of high HIV prevalence could lead to an increased risk of HIV-1 acquisition in the vaccinated population. The increased risk of HIV-1 acquisition appeared to be limited to men; a similar increase in risk was not seen in women in the Phambili trial.
Several follow-up studies suggested the potential mechanism for this increased susceptibility to HIV infection among men. The vaccine was highly immunogenic in the induction of HIV-specific CD4 and CD8 T cells; however, there was no difference in the frequency of T-cell responses after vaccination in men who did and did not later become infected with HIV in the Step Study.These findings suggest that immune responses induced by the HIV-specific vaccine were not the mechanism of increased acquisition. Participants with high frequencies of preimmunisation Ad5-specific T cells were associated with a decreased magnitude of HIV-specific CD4 responses and recipients of the vaccine had a decreased breadth of HIV-specific CD8 responses,suggesting that pre-existing Ad5 immunity might dampen desired vaccine-induced responses. Additional exploratory studies suggest that Ad5 immune complexes activate the dendritic cell–T cell axis, which might enhance HIV-1 replication in CD4 T cells.
Additionally, Ad5-specific CD4 T cells could have an increased susceptibility to HIV infection.In a non-human primate challenge study, infecting rhesus macaques with Ad5 and then immunising them with a replication-incompetent Simian immunodeficiency virus (SIV) vaccine based on Ad5 increased the risk of SIV acquisition from low-dose SIV penile challenge.Furthermore, subclinical inflammation and alterations to epithelial barriers at the inner foreskin, including increased densities of CCR5-positive CD4 T cells could contribute to high rates of sexually transmitted infections, including HIV, in uncircumcised men.
On the basis of these findings, we are concerned that use of an Ad5 vector for immunisation against severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) could similarly increase the risk of HIV-1 acquisition among men who receive the vaccine. Both the HIV and COVID-19 pandemics disproportionately affect vulnerable populations globally. Roll-out of an effective SARS-CoV-2 vaccine globally could be given to populations at risk of HIV infection, which could potentially increase their risk of HIV-1 acquisition.
This important safety consideration should be thoroughly evaluated before further development of Ad5 vaccines for SARS-CoV-2, and informed consent documents of these potential risks should reflect the considerable literature on HIV-1 acquisition with Ad5 vectors.

Understanding the emergence of novel viruses requires an accurate and comprehensive annotation of their genomes.
Overlapping genes (OLGs) are common in viruses and have been associated with pandemics but are still widely overlooked. We identify and characterize ORF3d, a novel OLG in SARS-CoV-2 that is also present in Guangxi pangolin-CoVs but not other closely related pangolin-CoVs or bat-CoVs. We then document evidence of ORF3d translation, characterize its protein sequence, and conduct an evolutionary analysis at three levels: between taxa (21 members of Severe acute respiratory syndrome-related coronavirus), between human hosts (3978 SARS-CoV-2 consensus sequences), and within human hosts (401 deeply sequenced SARS-CoV-2 samples). ORF3d has been independently identified and shown to elicit a strong antibody response in COVID-19 patients. However, it has been misclassified as the unrelated gene ORF3b, leading to confusion.
Our results liken ORF3d to other accessory genes in emerging viruses and highlight the importance of OLGs.


Compared to adults, children with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) have predominantly mild or asymptomatic infections, but the underlying immunological differences remain unclear. Here, we describe clinical features, virology, longitudinal cellular, and cytokine immune profile, SARS-CoV-2-specific serology and salivary antibody responses in a family of two parents with PCR-confirmed symptomatic SARS-CoV-2 infection and their three children, who tested repeatedly SARS-CoV-2 PCR negative. Cellular immune profiles and cytokine responses of all children are similar to their parents at all timepoints. All family members have salivary anti-SARS-CoV-2 antibodies detected, predominantly IgA, that coincide with symptom resolution in 3 of 4 symptomatic members. Plasma from both parents and one child have IgG antibody against the S1 protein and virus-neutralizing activity detected. Using a systems serology approach, we demonstrate higher levels of SARS-CoV-2-specific antibody features of these family members compared to healthy controls. These data indicate that children can mount an immune response to SARS-CoV-2 without virological confirmation of infection, raising the possibility that immunity in children can prevent the establishment of SARS-CoV-2 infection. Relying on routine virological and serological testing may not identify exposed children, with implications for epidemiological and clinical studies across the life-span.

I will post a short summary of Professor Luc Montagnier's video lecture on DNA emitting frequency later.



Dagobah Resident
Professor Luc Montagnier reveals that DNA emits frequencies.
 Since 2005, he has developed a new biophysical technology that detects the electromagnetic waves induced in water by the DNA of pathogenic bacteria and viruses and in the blood of patients suffering from chronic degenerative diseases. He also explains how oxidative stress caused by environmental pollution of electromagnetic waves attacks the molecules of our body which can be the cause of a number of diseases.
This film provides us with the fascinating testimony of a great scientist convinced of the benefits of preventive medicine.
Fréquences électromagnétiques de l’ADN 
par le Pr Luc Montagnier
"DNA emits low frequency emissions."

"Different waves depending on whether they come from bacteria or viruses."

"Biology has established that we emit waves."

"We need a revolution in biology that takes into account the progress of physics."

"From a structure of water, stored information that comes from DNA, genetic information.
We can reproduce this DNA by putting enzymes capable of reading this water structure."

"The electro-magnetic background noise of interference waves."

Nobel Prize Winner Takes Homeopathy Seriously


Dagobah Resident
Amazing water!
I found a video that is a must-see posted by mkrnhr.

Homeopathy as a science.

Kary Mullis, will certainly be happier with the use of his invention (PCR Test) in this context.

Water is the key element of life, but new information is coming to light on the element which we thought we were so familiar with: information which could potentially reimagine our tree of life. That is the belief shared by the advocates of a surprising theory called "water memory". For Prof. Luc Montagnier, water has the ability to reproduce the properties of any substance it once contained. Water would have the ability to retain a memory of the properties of the molecules. What if Alzheimer's, Parkinson's, autism, HIV and even cancer could be treated thanks to this controversial theory?


FOTCM Member
Hi Pierre,
I've taken some notes and links from some of the video lectures I've taken the time to watch (Thread ).
I haven't had time to dig deeper and connect the dots yet, but I thought that instead of keeping them until then, they might be useful to you in some way.
Thanks for sharing these interesting resources. As you will see, some are already integrated in "Mass extinction, evolutionary leap and the virus-information connection"

Actually, your post prompted me to share the book now in the thread, because, given the state of the world, I'm not sure we can finish it and publish it.

About 20 chapters are already written but not proofread, I hope the English is understandable. I will try to post the chapters one by one, regularly.

I will also post a new table of content and a new introduction, since these two parts got substantially modified since I last posted them.


Dagobah Resident
Yes Pierre, I imagine you did a lot of research as you did for your two previous books.
I thank you for your work and sharing.
And in the context of the world today, it's a great idea to post chapter by chapter, because I think it's important to keep a certain level of awareness and information to keep entropy low.
And that by posting your research earlier, you increase the level of awareness, thus maintaining entropy, which will finally allow you to publish your book, and Laura, hers on Caesar, in the right way.
ET voilà, que demande le peuple! (And that's it, what do the people want!)

And as all work deserves to be paid, I propose that those who will read chapter after chapter,
make a "special stone to the building" donation/ (un don "spécial Pierre à l'édifice"). :headbanger:


FOTCM Member

Table of content​

Table of content. 2

Introduction. 5

Part I : Comets and Mass Extinctions. 9

Chapter 1: Mass Extinctions. 10

Chapter 2: Causes of Mass Extinctions. 14

Chapter 3: The 27 million year cometary cycle. 20

Part II: Life Explosions. 31

Chapter 4: Peculiar Life-Forms around Cometary Impacts. 32

Chapter 5: The K/T Boundary. 38

Chapter 6: The Cambrian Life Explosion. 41

Chapter 7: Other Life Explosions. 45

Chapter 8: Saltationism vs Darwinism.. 54

Part III: Viruses are the Drivers of Life. 60

Chapter 9: The Enigma of Speciation. 61

Chapter 10: Species-Specific Eradication or Enhancement. 75

Chapter 11: Viruses Contribute to Mass Extinctions. 79

1/ Past virus-induced extinction. 79

2/ Recent virus-induced extinction. 82

3/ Are Viruses extinction agents past and present?. 83

Chapter 12: Anteriority of Viruses. 86

Chapter 13: Pervasiveness of Viruses. 90

Chapter 14: Beneficial Viruses. 96

Exogenous viruses. 97

Endoretroviruses: 100

Chapter 15: Terrain or germs?. 105

Part IV: Viruses and Meteorites. 113

Chapter 16: Comets and Plagues, an Historical Perspective. 114

Chapter 17: The Plague of Justinian. 117

Fatality and duration. 117

Origin. 118

Chapter 18: The Black Death. 122

Fatality and duration. 122

Pathogenic agent. 124

Origin. 127

Chapter 19: Society before and after both plagues. 132

Social conditions before both plagues. 132

Social and civilizational conditions after the plagues. 135

Two plagues, one pattern? 140

Chapter 20: Meteorites Carrying Microorganisms. 142

Chapter 21: Upper Atmosphere Microorganisms. 147

Chapter 22: Electromagnetic Pulses. 149

Chapter 23: Panspermia vs. Spontaneous Generation. 157

Part V: The information Field. 159

Chapter: Information Theory & consciousness. 160

Chapter: The Field. 167

Chapter: Collective resonance. 177

Part VI: Information Field, Viruses, DNA and Proteins. 182

Chapter: DNA as Antennas. 183

Chapter: Proteins as Tuners. 185

Chapter: DNA and Proteins Connect us to the Information Field. 193

Chapter: From Geometric Shapes to Electromagnetic Signatures. 196

Chapter: Resonance Cavities. 198

Chapter: The Interesting Case of Homeopathy. 206

Chapter: Fractal Transmission Increases Informational Depth. 211

Chapter: Delayed Life Explosion or Immediate One?. 221

Deleterous effects of raditions. 229

Pathogenesis and healing as information directed processes: 230

Part VII: 6th Extinction and Coronavirus. 232

Chapter: The ongoing 6th extinction. 233

Chapter: Authorities and Life. 235

Chapter: Authorities and Control 238

Chapter: Dumbing Down the Population. 240

Chapter: The Mind-Altering Virus. 242

Chapter: Man-Made Viruses. 243

Chapter: Man-Made SARS-CoV-2. 244

Chapter: Engineered for What Purpose?. 247

Chapter: Fort Detrick And Viruses. 249

Chapter: Fort Detrick and Mind Control 252

Chapter: Made in China or Made in the USA?. 254

Chapter: From an Obedience Vaccine to a Mutated Disobedience Virus?. 258

Chapter: Lock-down to Minimize Spread Not Fatality. 261

Chapter: Eugenics 2.0. 264

Chapter: The real cause of the 6th extinction. 275

Chapter: Conclusion. 283


FOTCM Member


In Earth Changes and the Human Cosmic connection, we saw that life on Earth is punctuated by mass extinctions, most them being due to cyclical cometary bombardments[1]. Besides the 28 million years cometary cycle that coincides with more than half the mass extinctions, virtually all mass extinctions seem to have been caused, at least partly, by cometary bombardments. In addition, all the other causes usually invoked for explaining mass extinctions whether volcanism, global cooling or plate tectonics are known consequences of cometary events. The above is the line of force of Part I: “Comets and Mass extinction”.

Comets are agents of mass destruction. And widely considered as such by science. But is it the whole story? While writing a second book titled “Cometary Encounters[2], I researched further the Tunguska overhead explosion and couldn’t help to notice some strange effects induced by the event. For example Tunguska trees revealed a growth rate multiplied by four in some cases.[3]

An increased growth doesn’t sound like a random detrimental mutation but like a beneficial one. Could cometary mass extinctions also improve life forms? Maybe Tunguska was an isolated case, so I had a look at other comet induced mass extinctions, in particular the well-documented K/T boundary[4] when 66 million years ago, the Chicxulub impact wiped out the dinosaurs along with 75%[5] of all species on the planet.

Right below the K/T boundary lie the fossils of the dinosaurs and right above the K/T boundary – meaning right after the mass extinction – lay the fossils of fully developed new species having superior complexity and no known ancestors[6].

The K/T boundary is not an oddity. Virtually each geological period exhibits the same pattern. It starts with a mass extinction quickly followed by new fully developed life forms appearing out of thin air. The correlation between the aftermath of mass extinction and new life-forms is stunning. In fact almost all major living branches of life appeared right after a mass extinction.

It suggests that major cometary impacts are not only acts of destruction through the removal of obsolete life forms during mass extinctions but also creative acts through the apparition of more elaborate life-forms. That’s the main idea developed in Part II: “Life explosion

Logically the next question was “How can cometary impact infuse new and more complex life-forms?” This question hovered for a while until the COVID hysteria, about which I wrote two articles[7]. During this process I learnt more about viruses, including their pervasive presence on the planet in general and in the genetic code of all life-forms, including humans in particular[8].

Not only viral DNA constitutes a large part of our genetic code but it has been demonstrated that this “foreign” DNA offers new beneficial functions to the host[9]. In the light of the above, viruses seem more fundamental than even life itself; they are the information carriers -genetic codes- from which biological life stems. Viruses are more than life: they are the very source of life; they are the informational precursors, the initiators of life forms. These, in a nutshell, are the main ideas presented in Part III “Viruses, Drivers of Life”.

Knowing the central role played by virus in the genetic code of life-forms, I started to wonder if it could be comet-borne viruses that triggered the evolutionary leaps that mark the aftermath of comet-induced mass extinctions.

The fact is that cometary material is unexpectedly rich in organic material.[10] More than that, several researchers including NASA’s head of astrobiology research Richard Hoover[11] have published a number of papers about the presence of microorganisms in meteoritic material. Not only meteorites carry microbes but the Earth’s upper atmosphere is also laden by microorganisms. Dozens of types of microorganisms, including three species[12] –two bacterial and one fungal- that are unknown on Earth have been found as high as 41 km[13], where no air from lower down would normally be transported[14]

So, meteorites directly carrying new viruses and meteorites puncturing the atmosphere and enabling the entrance of new viruses are the two non-mutually exclusive hypotheses to explain the infusion of new virus on Earth. This association between new viruses and cometary impacts is the topic addressed in part IV : “Viruses and Meteorites

At this point one obvious question emerged. How does viral DNA manage to shape new complex life forms? Mainstream science postulates that DNA is the code that controls the synthesis of proteins – the building blocks of life. Different DNA = different code = different proteins = different morphology. It’s a simple and neat process, but is it true?

How can this process explain that in one organism every single cell carries the same DNA, however these cells follow different developmental paths. One becomes muscle cell, another one a neuron, a third one a bone cell. How to explain that when a salamander loses a limb, it grows a perfect replica of it? Where is template, where is the blueprint of the whole limb? [15]. Given that the cells have the same DNA, it must something else that guides their differentiated and harmonious development.

I dedicated the last part[16] of Earth Changes and the Human Cosmic connection to demonstrating the existence of a non-local information field, including morphology-related information. I won’t transcribe here this 100-page part, but one excerpt might suffice to convey the main idea:

There’s a young student at this university who has an IQ of 126, has gained a first-class honors degree in mathematics, and is socially completely normal. And yet the boy has virtually no brain [...]When we did a brain scan on him, we saw that instead of the normal 4.5-centimeter thickness of brain tissue between the ventricles and the cortical surface, there was just a thin layer of mantle measuring a millimeter or so. His cranium is filled mainly with cerebrospinal fluid.[17]

An individual virtually devoid of any brain and being fully functional intellectually and socially suggest that our ideas, thoughts, memories might be non-local instead of stored/created in our brain as usually assumed.

That’s the main premise of the concept of information field that contains all information including the ones relating to life-forms morphology. This is the line of force structuring Part V: “The information Field”

If information, including morphic information is non-local, and the acquisition of new viral DNA enables morphologically more complex life-forms, so viral DNA must be able somehow to modulate the connection of life-forms to the Information Field.

Could viruses act like some antenna? Viruses are DNA and DNA recently revealed all the feature of a fractal antenna when exposed to electromagnetic fields:

The wide frequency range of interaction with EMF is the functional characteristic of a fractal antenna, and DNA appears to possess the two structural characteristics of fractal antennas, electronic conduction and self-symmetry. These properties contribute to greater reactivity of DNA with EMF in the environment,[18]

Viruses can modify DNA. DNA acts as an antenna connected to the Information Field. As will see, DNA also codes for proteins that act as a tuner to specific information within the Information Field. We will also see the geomagnetic conditions surrounding our planet alter the very expression of DNA, suggesting that it is combination of virally altered DNA and specific geomagnetic conditions that defines our connection the information field. These are main ideas suggested and developed in Part VI: Information Field, Viruses, DNA and Proteins.

At this point a few questions were left, among which: will we have the same fate as the dinosaurs? Will the survivors constitute the initiators of a new species? Unlike the previous parts of this book, this part is prospective. But one thing sure, all the ingredients are present for a 6th extinction to occur: the cyclical nature of mass extinctions, the 6th extinction that has already started, the human immunity that is weaker than ever, the ever increasing fireball activity and of course the tampering of DNA via “vaccines”. These topics and these questions will be addressed in Part VII: 6th Extinction and Coronavirus.

And now that you had an outlook on the content of this books, with further due, let’s jump in.

[1] Lescaudron, 2014. Chapter 15 “Enter Nemesis“.
[2] Lescaudron, Pierre. (2020). “Cometary Encounters”. Red Pill Press
[3] See: . Longo, G. et al. (1994). “Search for microremnants of the Tunguska cosmic body”. Planetary and Space Science. n. 2, 163--177.
Serra, R. et al. (1994). “Experimental hints on the fragmentation of the Tunguska cosmic body”. Planetary and Space Science, n. 9, 777--783.
[4] Cretaceous/Tertiary
[5] Kaiho, K. et al. (2016). “Global climate change driven by soot at the K-Pg boundary as the cause of the mass extinction”. Sci Rep 6, 28427
[6] Felix, Robert. (2008). “Magnetic Reversals and Evolutionary Leaps: The True Origin of Species”. Sugarhouse Publications. P. 33.
[7] Lescaudron, Pierre. (2020). “Compelling evidence that SARS-CoV-2 was manmade”.
Lescaudron, Pierre. (2021). “The Inanity of RNA Vaccines For COVID-19”.
[8] Moelling, K. & Broecker, F. (2019). “Viruses and Evolution - Viruses First? A Personal Perspective”. Frontiers in microbiology, 10, 523.
[9] Ryan, Frank. (2013). “Virolution“. Harper Collins UK.
[10] Cronin, J.R. et al. (1983). “Amino acids in meteorites”. Advances in Space Research. Vol. 3-9, Pages 5-18.
[11] See chapter “Impact On Human Populations”.
[12] Shivaji, S. et al. (2009). "Janibacter hoylei sp. nov., Bacillus isronensis sp. nov. and Bacillus aryabhattai sp. nov., isolated from cryotubes used for collecting air from upper atmosphere". IJSE Microbiology. 59 (Pt 12): 2977–86.
[13] 25 Mile
[14] Narlikar, JV. et al. (2003). "Balloon experiment to detect micro-organisms in the outer space". Astrophys Space Sci. 285 (2): 555–62
[15] See for example: Sheldrake, Rupert. (2009). “Morphic Resonance: The Nature of Formative Causation”. Inner Traditions/Bear.
[16] “Part IV: Role of the elites, the human-cosmic connection” pp.192-290.
[17] Lewin, Roger (1980). “Is your brain really necessary?”, Science, Dec 12, Vol.210, p.1232(3)
[18] Blank M., Goodman R. (2011). “DNA is a fractal antenna in electromagnetic fields”. Int J Radiat Biol. 87(4):409-15.
Personally, I am not a man of knowledge and I admit that these recent interventions, or your articles and book, Pierre (which I still haven't finished) are fascinating, although I unfortunately don't have the level to appreciate them at their true value, but there is a link that I have difficulty in establishing concerning mass extinction, and it is the following: I concieve the influence of cosmological events on living things, and whether they are cometary events, cosmic particles or even radiation or vibrations, they are part of what I would call natural events. But we are also in a particular period where eugenics is also very much at work and on a very large scale. I see the two happening together, and as much as for the firts one (which can perhaps be considered as an effect of the wave), I am tempted to offer the slightest resistance, as much, for the other, I am naturally inclined to offer a determined resistance.
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